Abstract
Introduction: Is cancer caused by cell degeneration or may it be that tumor formation in such cases may depend on various forms of plant infestation (i.e. mycoses)? Our previous research and its findings have shown that microbiological growth occurs in the calluses that occur in mammal tissues associated with cancer diseases. Other researchers have also observed non-human growth in cancer samples, but they have assumed that the samples must have been contaminated. Materials and method: A hard lump in one of a female patient's breasts was discovered. The diagnose breast cancer was determined and the ill breast was removed surgically. The tumor found during surgery was divided into six samples that were prepared for microscopy. Results: The microscopy revealed structures in the samples that were consistent with identical structures found in Ascomycota (spore-sac fungus). The probability that the structures found in this study would not be constituted by an Ascomycota is miniscule. Conclusion: The morphological structures in the samples in this study are fully consistent with the characteristic features of the Ascomycota. The illustrated findings thus show that cancer can be fungal growth. It is therefore necessary to widen cancer research and the paradigm it is based upon and involve mycologists in cancer research.

abstract
Clinical and experimental data suggest an association between the presence of bacterial and/or fungal infection and the development of different types of cancer, independently of chemotherapy-induced leukopenia. This has also been postulated for the development of lung cancer, however the prevalence and the exact species of the bacteria and fungi implicated, have not yet been described.
Conclusion
A diversity of pathogens could be identified in surgically extracted tissue samples of patients with lung cancer, with mycoplasma strains being present in all samples. These results point to an etiologic role for chronic infection in lung carcinogenesis. Confirmation of these observations and additional studies are needed to further characterize the etiologic role of inflammation in lung carcinogenesis, thus making it possible to apply new therapeutic modalities.